673 White cell telomere length and risk of premature myocardial infarction

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White cell telomere length and risk of premature myocardial infarction.

OBJECTIVE Biological age may be distinct from chronological age and contribute to the pathogenesis of age-related diseases. Mean telomeres lengths provide an assessment of biological age with shorter telomeres, indicating increased biological age. We investigated whether subjects with premature myocardial infarction (MI) had shorter leukocyte telomeres. METHODS AND RESULTS Mean terminal restr...

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Integrin Beta-3 Gene Polymorphism and Risk for Myocardial Infarction in Premature Coronary Disease

Background: Contradictory results have been obtained regarding the role of integrin, beta 3 (ITGB3) gene polymorphisms in occurrence of myocardial infarction (MI). Objectives: We aimed to assess the association between 1565C/T polymorphism of ITGB3 gene and increased risk for acute MI in patients with premature coronary artery disease (CAD). <strong...

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Telomere Length Variation in Juvenile Acute Myocardial Infarction

Leukocyte telomere length (LTL) provides a potential marker of biological age, closely related to the endothelial dysfunction and consequently to the atherosclerotic process. To investigate the relationship between the LTL and the risk of premature acute myocardial infarction and to evaluate the predictive value of LTL on the onset of major cardiovascular events, 199 patients from 18 to 48 year...

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eNOS Gene Variants and the Risk of Premature Myocardial Infarction

BACKGROUND Endothelial nitric oxide synthase (eNOS) as well as nitric oxide play an important role in the regulation of cardiovascular function. There are limited and controversial data regarding the impact of polymorphisms of eNOS gene that is implicated in the vasoconstrictive properties of the endothelium in the pathogenesis of premature myocardial infarction (MI). OBJECTIVE We examined wh...

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Premature Myocardial Infarction in Males

Background Several studies indicate that endogenous hormones play a role in the etiology of coronary artery disease, either as independent risk factors or indirectly, via an effect on lipids, lipoproteins, or other heart disease risk factors. Methods and Results The relation between endogenous hormone levels and premature (<56-year-old patients) myocardial infarction was assessed in a retrospec...

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ژورنال

عنوان ژورنال: European Heart Journal

سال: 2003

ISSN: 0195-668X

DOI: 10.1016/s0195-668x(03)94107-8